Restrictive loss of plakoglobin in cardiomyocytes leads to arrhythmogenic cardiomyopathy

Keyphrases

• Cardiomyocytes 100%
• Arrhythmogenic Right Ventricular Cardiomyopathy 100%
• Plakoglobin 100%
• Arrhythmogenic Cardiomyopathy 100%
• Ventricular Arrhythmia 50%
• Adherens Junction 50%
• Myocardium 33%
• Catenin 33%
• Cardiomyopathy 33%
• Mutant Mice 33%
• Desmosome 33%
• Clinical Manifestations 16%
• Therapeutic Intervention 16%
• Cell Death 16%
• Transforming Growth Factor-β 16%
• Gain-of-function mutation 16%
• Fibrosis 16%
• Dropout 16%
• Potential Therapeutics 16%
• Major Components 16%
• Ablating 16%
• Ultrastructural Analysis 16%
• Pathogenic Pathways 16%
• Cardiac Fibrosis 16%
• Ventricular Dilation 16%
• Cardiac Dysfunction 16%
• Naxos 16%
• Cutaneous Disorders 16%
• Gap Junction 16%
• Naxos Disease 16%
• Ventricular Aneurysm 16%
• Fibrofatty Replacement 16%
• Myocardial Disorder 16%

Medicine and Dentistry

• Cardiac Muscle Cell 100%
• Arrhythmogenic Right Ventricular Dysplasia 100%
• Plakoglobin 100%
• Myocardial Disease 100%
• Heart Ventricle Arrhythmia 42%
• Adherens Junction 42%
• Myocardium 28%
• Catenin 28%
• Desmosome 28%
• Diseases 28%
• Cell Death 14%
• Fibrosis 14%
• Transforming Growth Factor Beta 14%
• Cardiac Fibrosis 14%
• Loss of Function Mutation 14%
• Gap Junction 14%
• Aneurysm 14%

Biochemistry, Genetics and Molecular Biology

• Cardiac Muscle Cell 100%
• Plakoglobin 100%
• Cardiac Dysrhythmia 75%
• Adherens Junction 75%
• Catenin 50%
• Desmosome 50%
• Mouse Mutant 50%
• Cell Death 25%
• Transforming Growth Factor Beta 25%
• Loss of Function Mutation 25%