Recurrent Transient Ischemic Attack Induces Neural Cytoskeleton Modification and Gliosis in an Experimental Model

Keyphrases

• Gliosis 100%
• Transient Ischemic Attack 100%
• Cytoskeleton Modification 100%
• Neuronal Cytoskeleton 25%
• Neuroinflammation 16%
• Glial Fibrillary Acidic Protein 16%
• Neurofilament Light (NF-L) 16%
• Related Dementia 16%
• Ionized Calcium-binding Adapter Molecule 1 16%
• Apoptosis 8%
• High Risk 8%
• Astrocytes 8%
• Astrogliosis 8%
• Alzheimer's Disease 8%
• Cell Death 8%
• Cognitive Impairment 8%
• Tau Hyperphosphorylation 8%
• Cognitive Dementia 8%
• Transient Middle Cerebral Artery Occlusion (tMCAO) 8%
• Microtubule-associated Protein 2 (MAP2) 8%
• Microglia 8%
• Cerebral Cortex 8%
• Alzheimer's Dementia 8%
• β-site APP-cleaving Enzyme 1 (BACE1) 8%
• Pathological Changes 8%
• White Matter 8%
• Basal Ganglia 8%
• Corpus Callosum 8%
• Subacute Stage 8%
• Subsequent Stroke 8%
• AD Dementia 8%
• Disease-related Biomarker 8%
• Acute Stage 8%
• Microgliosis 8%
• Permanent Neurological Deficits 8%
• Attack Model 8%
• Neuropathophysiology 8%
• Vascular Contributions 8%

Neuroscience

• Cytoskeleton 100%
• Gliosis 100%
• Transient Ischemic Attack 100%
• Alzheimer's Disease 16%
• Glial Fibrillary Acidic Protein 16%
• Calcium Binding 16%
• Neurofilament Light Chain 16%
• Astrocyte 8%
• Astrogliosis 8%
• Cell Death 8%
• Hyperphosphorylation 8%
• Cognitive Disorders 8%
• Middle Cerebral Artery 8%
• Microglia 8%
• Microtubule Associated Protein 2 8%
• Cerebral Cortex 8%
• Basal Ganglia 8%
• Corpus Callosum 8%
• Cerebrovascular Accident 8%