Mouse HSF1 disruption perturbs redox state and increases mitochondrial oxidative stress in kidney

Keyphrases

• Kidney 100%
• Mitochondrial Oxidative Stress 100%
• Heat Shock Factor 1 (HSF1) 100%
• Redox State 100%
• Heat Shock Proteins 50%
• Oxidative Stress 25%
• Wild Mice 25%
• Physiological Condition 25%
• Superoxide Production 25%
• Heat Shock Protein 90 (Hsp90) 25%
• αB-crystallin 25%
• Hsp25 25%
• Mitochondrial Superoxide 25%
• Protective Effect 12%
• Protein Expression 12%
• Mitochondrial Membrane Potential 12%
• Ischemia-reperfusion Injury 12%
• Redox Status 12%
• Homeostasis 12%
• Mitochondrial Permeability Transition Pore Opening 12%
• Renal Cell 12%
• Pathophysiological Conditions 12%
• Crystallin Proteins 12%
• Cellular Redox Status 12%
• Heat Shock Protein 70 (HSP70) 12%
• HSP70 Gene 12%
• Membrane Potential Change 12%

Biochemistry, Genetics and Molecular Biology

• Oxidative Stress 100%
• Heat Shock Protein 100%
• HSF1 100%
• Heat Shock Transcription Factor 1 100%
• Wild Type Mouse 50%
• Hsp70 50%
• Hsp90 50%
• Crystallin 50%
• Downregulation 25%
• Transcription 25%
• Homeostasis 25%
• Protein Expression 25%
• Wild Type 25%
• Reperfusion 25%
• Mitochondrial Membrane Potential 25%
• Mitochondrial Permeability Transition Pore 25%

Pharmacology, Toxicology and Pharmaceutical Science

• Heat Shock Transcription Factor 1 100%
• Heat Shock Protein 100%
• Wild Type Mouse 50%
• Superoxide 50%
• Crystallin 50%
• Ischemia 25%
• Mitochondrial Permeability Transition Pore 25%