Involvement of p38 MAPK in reactive astrogliosis induced by ischemic stroke

Keyphrases

• Ischemic Stroke 100%
• P38 Mitogen-activated Protein Kinase (p38 MAPK) 100%
• Reactive Astrogliosis 100%
• Astrogliosis 57%
• Glial Fibrillary Acidic Protein 42%
• Mouse Model 28%
• Knock-in Mouse Model 28%
• Littermate 28%
• Protein Expression 14%
• Kinase Activation 14%
• No Significant Difference 14%
• Lesion Volume 14%
• Transient Middle Cerebral Artery Occlusion (tMCAO) 14%
• Neuronal Cells 14%
• Nervous System Diseases 14%
• Central Nervous System Injury 14%
• Non-neural 14%
• Molecular Trigger 14%
• Astrocyte Proliferation 14%
• Glial Scar Formation 14%
• Glia Scar 14%
• Middle Cerebral Artery Occlusion Model 14%
• Astrocytic 14%
• Astrocyte Migration 14%
• Scratch Injury 14%
• Motor Recovery 14%
• Scar Area 14%
• Primary Astrocyte Culture 14%
• Hypoxia Injury 14%

Neuroscience

• Brain Ischemia 100%
• Astrogliosis 100%
• P38 Mitogen-Activated Protein Kinases 100%
• Astrocyte 42%
• Glial Fibrillary Acidic Protein 42%
• Middle Cerebral Artery 28%
• Synaptophysin 28%
• In Vivo 14%
• In Vitro 14%
• Cell Signaling 14%
• Central Nervous System 14%
• Protein Expression 14%
• Glia 14%
• Nervous System Cell 14%