Intermittent hypoxia conditioning prevents behavioral deficit and brain oxidative stress in ethanol-withdrawn rats

Keyphrases

• Absorbance 12%
• Adaptation 12%
• Antioxidant 12%
• Behavioral Abnormalities 12%
• Behavioral Deficits 100%
• Behavioural Signs 12%
• Brain Oxidative Stress 100%
• Brain Regions 12%
• Breath Alcohol Concentration (BrAC) 12%
• Cerebellum Cortex 12%
• Chronic Ethanol Consumption 12%
• Dextrin 25%
• Ethanol 100%
• Ethanol Intake 12%
• Ethanol Intoxication 12%
• Ethanol Withdrawal 87%
• Hippocampus 12%
• Hypoxia 12%
• Intermittent Hypoxia 100%
• Intermittent Hypoxia Conditioning 100%
• Ischemic Injury 12%
• Male Rats 12%
• Mitochondrial Permeability Transition Pore Opening 12%
• N-acetylcysteine 12%
• Normoxia 12%
• Oxidative Damage 12%
• Oxidative Stress Proteins 12%
• Per Se 12%
• Permeability Transition Pore 25%
• Pore Opening 25%
• Protein Carbonyls 25%
• Protein Oxidation 25%
• Protein Permeability 12%
• Startle Response 12%
• Superoxide 37%
• Superoxide Production 12%
• Tissue Homogenate 12%
• Tremor 12%
• Withdrawal Signs 25%

Neuroscience

• Acetylcysteine 14%
• Alcohol Withdrawal Syndrome 100%
• Antioxidant 14%
• Cerebellum 14%
• Dextrin 28%
• Heat Shock Protein 14%
• Hippocampus 14%
• Mitochondrial Permeability Transition Pore 14%
• Oxidative Stress 100%
• Startle Response 14%
• Tremor 14%

Pharmacology, Toxicology and Pharmaceutical Science

• Acetylcysteine 10%
• Alcohol Intoxication 10%
• Alcohol Withdrawal Syndrome 70%
• Antioxidant 10%
• Dextrin 20%
• Heat Shock Protein 10%
• Hypoxia 10%
• Injury 10%
• Intermittent Hypoxia 100%
• Mitochondrial Permeability Transition Pore 10%
• Superoxide 40%