HIV-1 infected immune competent mononuclear phagocytes influence the pathways to neuronal demise

Keyphrases

• Human Immunodeficiency Virus Type 1 (HIV-1) 100%
• Mononuclear Phagocytes 100%
• Immune-competent 100%
• Monocyte-derived Macrophages 77%
• Activated Monocyte 55%
• Neuronal Injury 44%
• Secretory Products 22%
• Lipopolysaccharide 22%
• Soluble Factors 22%
• Culture Supernatant 22%
• Macrophage Culture 22%
• Apoptosis 11%
• Neuronal Damage 11%
• Immune Activation 11%
• Neuronal Apoptosis 11%
• Rat Neurons 11%
• Glutamate 11%
• Calcium Influx 11%
• Cell Death Pathways 11%
• N-methyl-D-aspartate Receptor (NMDAR) 11%
• Strain-dependent 11%
• Primary Neurons 11%
• Neuronal Signaling 11%
• Virion 11%
• Viral Replication 11%
• MK-801 11%
• Viral Strains 11%
• Dendritic Arbor 11%
• N-methyl-D-aspartate Receptor Antagonist 11%

Immunology and Microbiology

• Human Immunodeficiency Virus 1 100%
• Mononuclear Phagocyte 100%
• Macrophage 55%
• Supernatant 22%
• Macrophage Culture 22%
• Cell Death 11%
• Upregulation 11%
• Neuroapoptosis 11%
• Virion 11%
• Viral Replication 11%
• Virus Strain 11%
• Calcium Transport 11%
• Programmed Cell Death 11%

Neuroscience

• Human Immunodeficiency Virus 100%
• Mononuclear Phagocyte 100%
• Macrophage 77%
• Cell Death 11%
• Calcium Transport 11%
• Neuroapoptosis 11%
• Glutamic Acid 11%
• Aspartic Acid 11%
• NMDA Receptor Antagonist 11%
• Virus Strain 11%
• Receptor 11%
• Programmed Cell Death 11%