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Circulating Pro-Inflammatory Exosomes Worsen Stroke Outcomes in Aging
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Dive into the research topics of 'Circulating Pro-Inflammatory Exosomes Worsen Stroke Outcomes in Aging'. Together they form a unique fingerprint.
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Keyphrases
Aging
100%
Exosomes
100%
Stroke Outcome
100%
Serum Exosomes
100%
Aged Rats
44%
Synaptic Dysfunction
33%
Microglia
33%
Young Rats
33%
C3a Receptor
33%
Ischemic Stroke
22%
Inflammatory Mediators
22%
Systemic Inflammatory Response
22%
Phagoptosis
22%
Inflammatory Milieu
22%
Age-related
11%
Age-related Illnesses
11%
Systemic Administration
11%
Phagocytosis
11%
Age-related Decline
11%
Functional Integrity
11%
Proteomic Analysis
11%
Long-term Functional Outcome
11%
Brain Diseases
11%
CD46
11%
Complement Component 1q
11%
Sensorimotor Deficits
11%
Microglia Depletion
11%
Stroke Rehabilitation
11%
Receptor Inhibitors
11%
Vehicle Group
11%
Promising Therapy
11%
Neuroscience
Cerebrovascular Accident
100%
Exosome
100%
C3a Receptor
30%
Brain Ischemia
20%
Complement Component C3a
20%
Complement Component C3b
20%
Phagocytosis
10%
Proteomics
10%
CD46
10%
Complement Component C1q
10%
Complement Component C4b
10%
Brain Disease
10%
Synaptic Loss
10%
Immunology and Microbiology
Exosome
100%
Rat
90%
Mediator
20%
C3b
20%
Prevalence
10%
Phagocytosis
10%
CD46
10%
Proteomics
10%
Complement Component C1q
10%
C3a (Complement)
10%
Complement Component C4b
10%
Biochemistry, Genetics and Molecular Biology
Rat
100%
C3a Receptor
33%
Mediator
22%
C3b
22%
Prevalence
11%
Phagocytosis
11%
Proteomics
11%
CD46
11%