Basic sciences in clinical glaucoma: Steroids, ocular hypertension, and glaucoma

Keyphrases

• Antiglaucoma 25%
• Aqueous Humor Outflow 25%
• Aqueous Humor Outflow Resistance 25%
• Biochemical Changes 25%
• Causal Role 25%
• Cell Function 25%
• Cell Shape 25%
• Cell-to-cell 25%
• Cortisol Levels 25%
• Cortisol Metabolism 25%
• Cytoskeletal Organization 25%
• Elevated Intraocular Pressure 25%
• Extracellular Matrix Deposition 25%
• Glaucoma 100%
• Glaucoma Patients 25%
• Glucocorticoid Responsiveness 25%
• Individual Responsiveness 25%
• Intraocular Pressure 25%
• Molecular Mechanism 25%
• Morphological Changes 25%
• Ocular Administration 25%
• Ocular Hypertension 100%
• Primary Open-angle Glaucoma 100%
• Primates 25%
• Protein Expression 25%
• Route of Administration 25%
• Shape Function 25%
• Steroid-induced Ocular Hypertension 100%
• Systemic Administration 25%
• Therapeutic Implications 25%
• Trabecular Meshwork 75%
• Trabecular Meshwork Cells 25%
• Treatment Duration 25%

Neuroscience

• Aqueous Humor Outflow 16%
• Cat 8%
• Cell Function 8%
• Cortisol 16%
• Dog 8%
• Extracellular Matrix 8%
• Glaucoma 100%
• Glucocorticoid 100%
• Intraocular Pressure 16%
• Metabolic Pathway 8%
• Ocular Hypertension 100%
• Open-Angle Glaucoma 33%
• Protein Expression 8%
• Steroids 100%
• Trabecular Meshwork 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Cat 8%
• Dog 8%
• Glaucoma 100%
• Glucocorticoid 100%
• Hydrocortisone 16%
• Intraocular Hypertension 100%
• Leporidae 8%
• Open Angle Glaucoma 33%
• Pathogenesis 8%
• Route of Administration 8%
• Steroids 100%
• Systemic Administration 8%