Adoption Study of Obesity

Duane R. Bonds; Lon O. Crosby; William H. Dietz; Stanley M. Garn; Steven L. Gortmaker; Arthur Hartz; Alfred A. Rimm; Robert M. Hamer; James L. Levenson; Donald R. Miller; James R. Hebert; Albert J. Stunkard; Thorkild I.a. Sorensen; Craig Hanis; Thomas W. Teasdale; Ranajit Chakraborty; William J. Schull; Fini Schulsinger

doi:10.1056/NEJM198607103150211

Adoption Study of Obesity

Duane R. Bonds, Lon O. Crosby, William H. Dietz, Stanley M. Garn, Steven L. Gortmaker, Arthur Hartz, Alfred A. Rimm, Robert M. Hamer, James L. Levenson, Donald R. Miller, James R. Hebert, Albert J. Stunkard, Thorkild I.a. Sorensen, Craig Hanis, Thomas W. Teasdale, Ranajit Chakraborty, William J. Schull, Fini Schulsinger

School of Biomedical Sciences

Research output: Contribution to journal › Letter › peer-review

1 Scopus citations

Abstract

To the Editor: The observation of Stunkard et al. (Jan. 23 issue)¹ that obesity in offspring was more strongly correlated with maternal than paternal size may have an alternative explanation. There is a growing body of literature suggesting that offspring who receive increased amounts of glucose in utero may be different from those who do not.² ³ ⁴ ⁵ ⁶ Therefore, variations in the intrauterine environment, as opposed to genetics, could explain the observation of Stunkard et al. The offspring of women who were diabetic during pregnancy are different from those of women who became diabetic after pregnancy, in having an increased incidence of.

Original language	English
Pages (from-to)	128-130
Number of pages	3
Journal	New England Journal of Medicine
Volume	315
Issue number	2
DOIs	https://doi.org/10.1056/NEJM198607103150211
State	Published - 10 Jul 1986

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Bonds, D. R., Crosby, L. O., Dietz, W. H., Garn, S. M., Gortmaker, S. L., Hartz, A., Rimm, A. A., Hamer, R. M., Levenson, J. L., Miller, D. R., Hebert, J. R., Stunkard, A. J., Sorensen, T. I. A., Hanis, C., Teasdale, T. W., Chakraborty, R., Schull, W. J., & Schulsinger, F. (1986). Adoption Study of Obesity. New England Journal of Medicine, 315(2), 128-130. https://doi.org/10.1056/NEJM198607103150211

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title = "Adoption Study of Obesity",

abstract = "To the Editor: The observation of Stunkard et al. (Jan. 23 issue)1 that obesity in offspring was more strongly correlated with maternal than paternal size may have an alternative explanation. There is a growing body of literature suggesting that offspring who receive increased amounts of glucose in utero may be different from those who do not.2 3 4 5 6 Therefore, variations in the intrauterine environment, as opposed to genetics, could explain the observation of Stunkard et al. The offspring of women who were diabetic during pregnancy are different from those of women who became diabetic after pregnancy, in having an increased incidence of.",

author = "Bonds, {Duane R.} and Crosby, {Lon O.} and Dietz, {William H.} and Garn, {Stanley M.} and Gortmaker, {Steven L.} and Arthur Hartz and Rimm, {Alfred A.} and Hamer, {Robert M.} and Levenson, {James L.} and Miller, {Donald R.} and Hebert, {James R.} and Stunkard, {Albert J.} and Sorensen, {Thorkild I.a.} and Craig Hanis and Teasdale, {Thomas W.} and Ranajit Chakraborty and Schull, {William J.} and Fini Schulsinger",

year = "1986",

month = jul,

day = "10",

doi = "10.1056/NEJM198607103150211",

language = "English",

volume = "315",

pages = "128--130",

journal = "New England Journal of Medicine",

issn = "0028-4793",

publisher = "Massachussetts Medical Society",

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TY - JOUR

T1 - Adoption Study of Obesity

AU - Bonds, Duane R.

AU - Crosby, Lon O.

AU - Dietz, William H.

AU - Garn, Stanley M.

AU - Gortmaker, Steven L.

AU - Hartz, Arthur

AU - Rimm, Alfred A.

AU - Hamer, Robert M.

AU - Levenson, James L.

AU - Miller, Donald R.

AU - Hebert, James R.

AU - Stunkard, Albert J.

AU - Sorensen, Thorkild I.a.

AU - Hanis, Craig

AU - Teasdale, Thomas W.

AU - Chakraborty, Ranajit

AU - Schull, William J.

AU - Schulsinger, Fini

PY - 1986/7/10

Y1 - 1986/7/10

N2 - To the Editor: The observation of Stunkard et al. (Jan. 23 issue)1 that obesity in offspring was more strongly correlated with maternal than paternal size may have an alternative explanation. There is a growing body of literature suggesting that offspring who receive increased amounts of glucose in utero may be different from those who do not.2 3 4 5 6 Therefore, variations in the intrauterine environment, as opposed to genetics, could explain the observation of Stunkard et al. The offspring of women who were diabetic during pregnancy are different from those of women who became diabetic after pregnancy, in having an increased incidence of.

AB - To the Editor: The observation of Stunkard et al. (Jan. 23 issue)1 that obesity in offspring was more strongly correlated with maternal than paternal size may have an alternative explanation. There is a growing body of literature suggesting that offspring who receive increased amounts of glucose in utero may be different from those who do not.2 3 4 5 6 Therefore, variations in the intrauterine environment, as opposed to genetics, could explain the observation of Stunkard et al. The offspring of women who were diabetic during pregnancy are different from those of women who became diabetic after pregnancy, in having an increased incidence of.

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U2 - 10.1056/NEJM198607103150211

DO - 10.1056/NEJM198607103150211

M3 - Letter

C2 - 3724799

AN - SCOPUS:0022579389

SN - 0028-4793

VL - 315

SP - 128

EP - 130

JO - New England Journal of Medicine

JF - New England Journal of Medicine

IS - 2

ER -

Adoption Study of Obesity

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