Adoption Study of Obesity

Duane R. Bonds, Lon O. Crosby, William H. Dietz, Stanley M. Garn, Steven L. Gortmaker, Arthur Hartz, Alfred A. Rimm, Robert M. Hamer, James L. Levenson, Donald R. Miller, James R. Hebert, Albert J. Stunkard, Thorkild I.a. Sorensen, Craig Hanis, Thomas W. Teasdale, Ranajit Chakraborty, William J. Schull, Fini Schulsinger

Research output: Contribution to journalLetterpeer-review

1 Scopus citations


To the Editor: The observation of Stunkard et al. (Jan. 23 issue)1 that obesity in offspring was more strongly correlated with maternal than paternal size may have an alternative explanation. There is a growing body of literature suggesting that offspring who receive increased amounts of glucose in utero may be different from those who do not.2 3 4 5 6 Therefore, variations in the intrauterine environment, as opposed to genetics, could explain the observation of Stunkard et al. The offspring of women who were diabetic during pregnancy are different from those of women who became diabetic after pregnancy, in having an increased incidence of.

Original languageEnglish
Pages (from-to)128-130
Number of pages3
JournalNew England Journal of Medicine
Issue number2
StatePublished - 10 Jul 1986


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