Aberrant histone acetylation promotes mitochondrial respiratory suppression in the brain of alcoholic rats

Keyphrases

• Alcoholic 100%
• Histone Acetylation 100%
• Respiratory Suppression 100%
• Let-7f 87%
• Ethanol Withdrawal 50%
• Trichostatin A 37%
• Ethanol 25%
• 2-cycles 25%
• Mitochondrial Respiration 25%
• Antagomir 25%
• Respirometry 25%
• Small Interfering RNA (siRNA) 12%
• Fold Increase 12%
• Gene Expression 12%
• Prefrontal Cortex 12%
• Co-treatment 12%
• Cell Death 12%
• Hippocampal Neurons 12%
• Mitochondria 12%
• Quantitative PCR 12%
• Neuronal Damage 12%
• Immunostaining 12%
• MicroRNA 12%
• Male Rats 12%
• Cell Viability 12%
• Control Diet 12%
• Abstaining 12%
• Calcein 12%
• Acetylation 12%
• Drinking Problems 12%
• TaqMan 12%
• HT22 12%
• Cellular Damage 12%
• Cell Integrity 12%
• Mitochondrial Damage 12%
• Chronic Ethanol 12%
• Multiple Episodes 12%
• Ethanol Exposure 12%
• Mitochondrial Integrity 12%
• Extracellular Flux 12%
• Mitochondrial Deficits 12%
• Histone Proteins 12%

Neuroscience

• Histone Acetylation 100%
• Alcohol Withdrawal Syndrome 50%
• Trichostatin A 37%
• Mitochondrial Respiration 25%
• Antagomir 25%
• Promoter Region 12%
• Gene Expression 12%
• Prefrontal Cortex 12%
• Cell Death 12%
• Immunohistochemistry 12%
• Real-Time Polymerase Chain Reaction 12%
• Mitochondrion 12%
• Cell Viability 12%
• MicroRNA 12%
• Cell Damage 12%
• Small Untranslated RNA 12%
• Acetylation 12%
• Histone 12%

Pharmacology, Toxicology and Pharmaceutical Science

• Histone 100%
• Alcohol Withdrawal Syndrome 44%
• Trichostatin A 33%
• Antagomir 22%
• Cell Viability 11%
• Calcein 11%
• microRNA 11%
• Small Untranslated RNA 11%